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Hydroxyurea Mechanism Of Action In Leukemia

Study of Dasatinib in Patients With Chronic Myelogenous Leukemia – Full Text View – nbsp; The goal of this clinical research study is to learn if BMS-354825 (dasatinib) can help to control CML in chronic phase. The safety of this drug will also be tter outcomes in patients withchronic myeloid leukemia: resultsNCT00254423 History of Changes Myeloid Leukemia, MyelogenousPositive Leukemia, Myeloid Molecular Mechanisms of Pharmacological Action Pharmacologic Elucidating critical mechanisms of deregulated stem cell turnover in the chronic phase of chronic nbsp; Leukemia is one of the leading journals in hematology and oncology. It is published monthly and covers all aspects of the research and treatment of leukemia and allied diseases. Studies of normal hemopoiesis arExpression of bcr-abl abrogatesfactor-dependent growth of human hematopoietican autocrine mechanism. Blood 1994biological defect in chronic myelogenous leukemia. Cancer ResInhibition of apoptosis byproduction and action of IL-3 and Elucidating critical mechanisms of deregulated stem cell turnover in the chronic phase of chronic nbsp; Leukemia is one of the leading journals in hematology and oncology. It is published monthly and covers all aspects of the research and treatment of leukemia and allied diseases. Studies of normal hemopoiesis arExpression of bcr-abl abrogatesfactor-dependent growth of human hematopoietican autocrine mechanism. Blood 1994biological defect in chronic myelogenous leukemia. Cancer ResInhibition of apoptosis byproduction and action of IL-3 and NTP Research Concept Template nbsp; Nomination Background and Rationale: Hydroxyurea was nominated to the NTP for toxicological testing by a private citizen and the NIEHS (based on the findings from the CERHR report- see below). hazard and risk. In addition, a better understanding of the mode of action and risk/benefit may help spur research in developing a betterMerrill, G. F. Hydroxyurea arrests DNA replication by a mechanism that preserves basal Guidelines for the Use of Antiretroviral Agents in HIV-1-Infected Adults and Adolescents December nbsp; How to Cite the Adult and Adolescent Guidelines: Panel on Antiretroviral Guidelines for Adults and Adolescents. Guidelines for the use of antiretroviral agents in HIV-1-infected adults and adolescents. Departmecombination therapy in 1996. New drugs haveapproved that offer new mechanisms of action, improvements in potency and activityincluding some with new mechanisms of action, this goal is now possible in many patients, including Combination Chemotherapy in Treating Patients With Untreated Acute Lymphoblastic Leukemia – Full nbsp; RATIONALE: Drugs used in chemotherapy use different ways to stop cancer cells from dividing so they stop growing or die. Combining more than one drug may kill more cancer ase II trial of dose intensification of Daunorubicin andintrathecal Methotrexate in place of cranial irradiationBertagnolli, Cancer and Leukemia Group B ClinicalTrialsAntimetabolites Molecular Mechanisms of Pharmacological Action Pharmacologic

Temsirolimus and Imatinib Mesylate in Treating Patients With Chronic Myelogenous Leukemia – Full nbsp;

doxorubicin-induced dna damage: Topics by nbsp; damage. In this study, we show that the activation of the MMR component hMLH1 in response to doxorubicin (DOX) treatment requires the presence of BRCA1 and that this phenomenon is mediated by an ATM/ATR dependeresults demonstrate a novel mechanism of action for sodium salicylatefurther study on the mechanism of topoisomerase II inhibitionPropofol induces DNA damage in mouse leukemic monocyteapoptosis in human and murine leukemia cell lines. Yet, whether Tipifarnib, Cytarabine, and Daunorubicin in Treating Older Patients With Acute Myeloid Leukemia – nbsp; RATIONALE: Tipifarnib may stop the growth of cancer cells by blocking some of the enzymes needed for cell growth. Drugs used in chemotherapy, such as cytarabine and daunorubicin, work in different ways to stop Additional relevant MeSH terms: Leukemia Leukemia, Myeloid, Acute Leukemia, Myeloid Neoplasms by HistologicAntineoplastic Antimetabolites Molecular Mechanisms of Pharmacological Action Pharmacologic Actions Antineoplastic Clofarabine, Cytarabine, and Idarubicin in Treating Patients With Intermediate-Risk or High-Risk nbsp; RATIONALE: Drugs used in chemotherapy, such as clofarabine, cytarabine, and idarubicin, work in different ways to stop the growth of cancer cells, either by killing the cells or by stopping them from dividing. chronic myelogenous leukemia in blast crisis orfor 14 days of hydroxyurea No CNS leukemia PATIENT CHARACTERISTICSMyeloid, Acute Leukemia, Myeloid MyelodysplasticAntimetabolites Molecular Mechanisms of Pharmacological Action Pharmacologic Study of Dasatinib (BMS-354825) in Patients With Accelerated Phase Chronic Myeloid Leukemia – Full nbsp; The purpose of this clinical research study is to learn if BMS-354825 will have activity, defined by hematologic response, in subjects who have accelerated phase chronic myeloid leukemia (CML) who are resistantin the treatment of chronic myeloid leukemia in accelerated phaseNCT00101647 History of Changes ObsoleteChronic myelogenous leukemia (CML): AcceleratedInhibitors Molecular Mechanisms of Pharmacological Action Pharmacologic Phase I/II Study of Decitabine and Valproic Acid in Relapsed/Refractory Leukemia or Myelodysplastic nbsp; Valproic acid is a medication that is currently used in the prevention of seizures, bipolar disorder, and migraine headaches. Researchers hope that it may improve the effects of decitabine. Decitabine is a chemPhase 1/2 study of the combination of 5-aza-2valproic acid in patients with leukemia. Blood. 2006NCT00075010 History of Changes OtherPharmacologic Actions Enzyme Inhibitors Molecular Mechanisms of Pharmacological Phase I/II Study of Decitabine and Valproic Acid in Relapsed/Refractory Leukemia or Myelodysplastic nbsp; Valproic acid is a medication that is currently used in the prevention of seizures, bipolar disorder, and migraine headaches. Researchers hope that it may improve the effects of decitabine. Decitabine is a chemPhase 1/2 study of the combination of 5-aza-2valproic acid in patients with leukemia. Blood. 2006NCT00075010 History of Changes OtherPharmacologic Actions Enzyme Inhibitors Molecular Mechanisms of Pharmacological

mouse monocytic cells: Topics by nbsp;

Interferon Alfa in Treating Patients With Newly Diagnosed Chronic Myelogenous Leukemia – Full Text nbsp; PURPOSE: Randomized phase III trial to compare the effectiveness of low-dose or high-dose interferon alfa in treating patients who have newly diagnosed chronic myelogenous leukemia. leukapheresis allowed Hydroxyurea may be startedcomparison of low-doseinterferon-alfa in chronic myeloid leukemia: prospective collaboration of 3 joint trialsInterferons Hydroxyurea AntiviralPharmacologic Actions Immunologic Molecular Mechanisms of Pharmacological cml chronic phase: Topics by nbsp; Note: This page contains sample records for the topic cml chronic phase from . While these samples are representative of the content of , they are not comprehensive nor are they the most cchronic myeloid leukemia PubMed Centralchronic myeloid leukemia (CML) hasbusulfan, hydroxyurea) to interferonwith IFN? in CML demonstratedpercentage of patients treatedcured. The mechanisms by which IFNactivation of leukemia-specificmechanisms of action. Nonetheless Hydroxyurea for the Treatment of Sickle Cell Disease Agency for Healthcare Research Quality nbsp; Topic page summarizing evidence report on hydroxyurea for the treatment of sickle cell d summaries of research findingsfindings on quality of and access toDates Grant Mechanisms DescriptionsSearchable database of AHRQ GrantsTransformation in Organizations and Networks II (ACTION II) ChildAssessments Hydroxyurea for the Treatment Combination Chemotherapy Followed by Peripheral Stem Cell Transplantation in Treating Patients With nbsp; RATIONALE: Drugs used in chemotherapy use different ways to stop cancer cells from dividing so they stop growing or die. Bone marrow transplantation may be able to replace immune cells that were destroyed by chrelevant MeSH terms: Leukemia Leukemia, Myeloid Leukemia, Myelogenous, Chronicphosphate Etoposide Hydroxyurea Mitoxantrone LenograstimAntimetabolites Molecular Mechanisms of Pharmacological Action Pharmacologic Actions Combination Chemotherapy Followed by Peripheral Stem Cell Transplantation in Treating Patients With nbsp; RATIONALE: Drugs used in chemotherapy use different ways to stop cancer cells from dividing so they stop growing or die. Bone marrow transplantation may be able to replace immune cells that were destroyed by chrelevant MeSH terms: Leukemia Leukemia, Myeloid Leukemia, Myelogenous, Chronicphosphate Etoposide Hydroxyurea Mitoxantrone LenograstimAntimetabolites Molecular Mechanisms of Pharmacological Action Pharmacologic Actions loss-of-function mechanisms contribute: Topics by nbsp; Note: This page contains sample records for the topic loss-of-function mechanisms contribute from . While these samples are representative of the content of , they are not comprehensive nounderstanding the mechanisms of radiation-induced leukemia/lymphoma development. In the present studySTRONTIUM AND CALCIUM IN BONE AND BLOODCONTRIBUTION TO THE MECHANISM OF ACTION OF PARATHYROIDCitations DatabaseIn rats containing

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